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Metformin 500 mg tab

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Glimepiride vs metformin

Clinical Care/Education/Nutrition, a randomized, single-blind comparative study, michael Gottschalk, MD 1, Thomas Danne, MD 2, Aleksandra Vlajnic, MD 3 and. Cara, MD 4 5 1University of California, San Diego Medical Center, San Diego, California 2Kinderkrankenhaus auf der Bult, Diabetes-Zentrum für Kinder und Jugendliche, Hannover, Germany 3Sanofi-Aventis.S., Bridgewater, New Jersey 4Children's Hospital of Michigan, Wayne State University, Detroit, Michigan 5Henry Ford Medical Center, Sterling Heights. Address correspondence and reprint requests to Michael Gottschalk, University of California, San Diego Medical Center, 3020 Childrens Way, MC 5103, San Diego, CA 92123. Diabetes Care 2007 Apr; 30(4 790-794. Previous, next, a randomized, single-blind comparative study, abstract. Objective, to compare the efficacy and safety of glimepiride versus metformin in glimepiride vs metformin pediatric subjects with type 2 diabetes inadequately controlled with diet and exercise alone or glimepiride vs metformin oral monotherapy. Research design AND methods, this 26-week, single-blind, active-controlled, multinational study randomized 285 subjects to receive glimepiride (18 mg once daily) or metformin (5001000 mg twice daily) for 24 weeks. The primary end point was mean change in A1C from baseline to week. Safety was assessed by incidence of hypoglycemia and other adverse events. Results, significant reductions from baseline A1C were seen in both the glimepiride (0.54,.001) glimepiride vs metformin and metformin (0.71,.0002) groups. A total.4 (56 of 132) and.1 (63 of 131) of subjects in the glimepiride and metformin groups, respectively, in the intent-to-treat population achieved A1C.0 at week. No significant differences were observed between groups in reductions in A1C and self-monitored blood glucose levels, changes in serum lipid concentrations, or hypoglycemia incidence. Significant differences were observed in mean changes from baseline in BMI between groups (0.26 kg/m2 for glimepiride and.33 kg/m2 for metformin;.003). The adjusted mean body weight increase was.97 kg for glimepiride and.55 kg for metformin (. A hypoglycemic episode with blood glucose 50 mg/dl (.8 mmol/l) was experienced.9 and.2 of glimepiride- and metformin-treated subjects, respectively. A single severe hypoglycemic event occurred in each group. Conclusions, glimepiride reduced A1C similarly to metformin with greater weight gain, and there was comparable safety over 24 weeks in the treatment of pediatric subjects with type 2 diabetes. The incidence of type 2 diabetes has been rapidly increasing in the pediatric population ( 1 ). Type 2 diabetes rarely was seen in pediatric subjects before the 1990s, but by 1999 estimates of new-onset type 2 diabetic cases in this population ranged from 8 to 45, depending on ethnicity and geographic location ( 1 ). This increase may, in part, be attributed to the dramatic rise in childhood obesity (now estimated at 1520 glimepiride vs metformin of the pediatric population). Further, increased intake of calories and dietary fats combined with inadequate physical activity may be contributing to the development of childhood obesity and type 2 diabetes ( 1, 2 ). As in adults, type 2 diabetes in children and adolescents results from both insulin resistance and relative pancreatic -cell secretory failure, with most subjects presenting with symptomatic hyperglycemia ( 2 ). This similarity, combined with the documented efficacy of oral antidiabetic therapy in adults, suggests that oral agents will be equally effective in children and adolescents ( 2 ). Type 2 diabetes may have an earlier and more aggressive course in pediatric patients ( 3, 4 therefore, they are likely to be at a higher risk for developing complications and need the best possible glycemic control in the early stage of their disease. Although use of oral antidiabetic agents in adults with type 2 diabetes is well established, much less is known about use in pediatric patients. Thus, efficacy and safety data in the pediatric population are urgently needed to meet the goals of improved glycemic control and long-term outcomes in type 2 diabetes. Metformin is a biguanide that suppresses basal hepatic glucose uptake and increases insulin-mediated glucose uptake in peripheral muscle. Because metformin does not stimulate insulin secretion, hypoglycemia is uncommon with monotherapy ( 5 making it an attractive agent for use in children and adolescents.

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Glimepiride vs metformin